Discussion Board

The way you talk is like you don't really understand how type 2 diabetes works and how it is not the same as insulin resistance.

if you have REAL diabetes, insulin resistant or not, it is because your pancreatic insulin output is limited compared to a non-diabetic.

Not true.

Type 1 Diabetes, is characterized by the bodies inability to produce insulin (or it is greatly diminished). This is why they must take insulin injections. This is Type 1.

However, insulin resistance, metabolic syndrome and Type 2 Diabetes is characterized by an increase in insulin output & levels. In response to the "resistance" the body secretes more insulin, not less. Testing insulin levels is one way to tell the difference. Type 2 Diabetics make plenty of insulin, it is just the body does not allow it to work properly. This is Type 2, IR &/or MS.

There is a third situation, where if Type 2 goes untreated (or poorly treated) for a very long time, the body may eventually fail to be able to keep up the demand for more insulin & output decreases. If true, they will most likely eventually need to take insulin, though I have seen even this reversed.

EESystem

Does the EES system cure bad plastic surgery?
f1jim

Ok. Let's go back to where you express some significant mistaken beliefs regarding diabetes. This is from several pages back in this thread, where Jeff Novick pointed out your mistakes.

You wrote:

Jeff Novick wrote:
Let's not keep repeating the same mistaken ideas with respect to diabetes.

If you have too much insulin and its not working that is simply insulin resistance.

Diabetes mellitus is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action, or both. Type 2 diabetes, the most prevalent form of the disease, is often asymptomatic in its early stages and can remain undiagnosed for many years.

Type 1 always has insufficient insulin but for T2 high blood glucose is the determining factor, insufficient insulin is optional.
[url]care.diabetesjournals.org/content/27/suppl_1/s11.full[/url]

That doesnt help when talking about things though does it. We need to be clear about what we are actually talking about.

I will not participate in this thread any further. Nobody seem interested in actually understanding anything.

Hate to be the curmudgeon but just a lot of big "scientific" sounding words saying nothing. This is just another in a continuing river of computer driven, flashy screen displays, that have no proof other than a high tech sounding explanation. Fast talk, smoke and mirrors. Clinical data is nice but is not controlled or blinded so results could very easily be placebo effect. Placebo effect is grand but no need for very expensive, high tech equipment for that, just meditate; it's free and you can do it even if the power goes out. EES may actually do something beyond that but there is so far no real proof of that. There are literally dozens of different but similar set-ups on the market. They come, they become a brief fad on the fringes, they go...

How does clarifying terms and definitions not help?

Bruce Lipton (a cell biologist) shares there are three things that impact a cell: trauma, toxins, thoughts. It seems Dr. Shintani is connecting with his Clients through all three. I feel he is tweaking his program and must be having some positive feedback that it works. I know you work as a acupuncturist and it might be worth your while to have a chamber. I have a feeling it works with our epigenetic system as consciously knowing subconsciously we eat the same amount of weight of food daily allows us to eat mindfully.

It does but that definition doesnt clarify anything. That is my point. It confuses the issue.

Maybe we should use the terms insulin deficiency and insulin resistance.

Sorry, nothing there that isn't likely placebo

My point, maybe should have been more explicit, is that if you continue to define T2 diabetes as insulin insufficiency, that tells the 90% or so for whom it is only IR that they can't be cured and will always need insulin or other drugs, but they can. It gets back to earlier in the thread about can it T2 be cured or only treated? If it is only IR in a particular case, then if you eliminate the IR the disease is gone, cured (unless/until they eat in a way to re-establish insulin resistance of course). For those who are T2 who in fact have insulin insufficiency, they will likely need some amount of medication for every though greatly reduced amount once they eliminate their IR.