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Join Dr. McDougall along with fellow McDougallers in lively discussions and share your opinions.
Maer wrote:As others have posted, I, too, wonder about incrurring long-term damage from sugar spikes; this happens with potatoes and more than about 1 c (cooked measure) of whole grains.
Has this changed your choice of what you eat?
Dan Rivera wrote:Has your expanded viewpoint changed your thoughts on intermittent fasting?
Dan Rivera wrote: While increased lipolysis sends fatty acids through the bloodstream, doesn't it do so during fasting periods for the fats to be oxidized and burned as energy (as also happens during fed states), and not to be deposited in places where fats shouldn't accumulate?
During fasting or starvation, free-fatty-acids are released during lipolysis into the liver and muscles to be burned as energy, this is called fat-oxidation. During the fed-state and especially while eating a starch-based-diet, fat-oxidation is inhibited and replaced with carbohydrate-oxidation, insulin is what mediates this shift. When carbohydrate-oxidation is taking place, fatty-acids are shuttled back and "locked away" in adipose-tissue... where they belong.
Dan Rivera wrote:If this were true, then why is fat still oxidized during the fed state?
Dan Rivera wrote:is the up regulation of the release of stored fat into the bloodstream during a fasted state necessarily harmful considering these fats are being burned, and not stored?
didi wrote:If fat is stored in the muscles on a high starch diet then how come so many people lose weight on a high carb diet? And if fat is stored in the liver, how come the liver numbers become normal which can be shown on a blood test, and those with fatty liver actually find that the fatty liver can be reversed?
I don't know if I am understanding this. If lipolysis is the breakdown of fats in the cells then how can that cause metabolic syndrome?
Dan Rivera wrote:Of note on the Atkins diet, fat intake often exceeds the rate at which they can be burned, and being a higher percentage of calories from fat, whether hypocaloric or in calorie balance, the total calories of fat deposited into fat cells far exceeds the rate at which occurs on a high carb low fat diet.
Dan Rivera wrote: To me it seems counter intuitive that going without food enable a disease like type 2, when in fact there is evidence that calorie restriction and fasting is beneficial.
It would also make little sense to me that the physiologically healthy release of fat into the bloodstream, such as in cases of weight loss, calorie restriction, or intermittent fasting, would support a disease like Type 2 diabetes.
misterE wrote:The thing is thou, on the Atkins-diet, fat is never transported into the adipose-tissue, it either floats in the blood or accumulates in ectopic-sites, due to the lack of insulin-secretion.
Actually studies show that during starvation and prolonged-fasting you actually become insulin-resistant, due to the increase of free-fatty-acids. Studies also show that rapid-weight-loss, prolonged-fasting and ketogenic-diets can actually induce NAFLD (non alcoholic fatty liver disease).
misterE wrote: The breakdown of body-fat (adipose-tissue) releases a huge amount of free-fatty-acids into the bloodstream, these free-fatty-acids then accumulate in non-adipose-tissues like the muscles or liver and block the ability of insulin to work properly. Eating starch (and stimulating insulin-secretion) shuttles these free-fatty-acids back into the adipose-tissue where they belongs.